A new study provides an answer for a long-standing scientific puzzle: why don't mice with CF gene mutations develop the life-threatening lung disease that affects most people with CF? The answer to the puzzle identifies a proton pump called ATP12A as a potential target for new CF therapies.
A new study provides an answer for a long-standing scientific puzzle: why don't mice with CF gene mutations develop the life-threatening lung disease that affects most people with CF? The answer to the puzzle identifies a proton pump called ATP12A as a potential target for new CF therapies.
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