SIDS: Serotonin neurons contribute to fail-safe mechanism that ensures recovery from interrupted breathing

Research in mice adds to evidence that dips in the activity of serotonin neurons may increase SIDS risk. The study reveals the activity of serotonin-producing neurons in the infant mouse brain contributes to a fail-safe switch that allows recovery from interrupted breathing. If replicated in human studies, findings could pave the way to screening for SIDS risk, new therapies.